This is a cross-section of the Chen-Lupino boy's myocardium.

这是Chen-Lupino心肌的横截面。

In this case, less blood and oxygen gets through the coronary artery to the heart tissue, which damages the myocardium.

在该情况下，冠状动脉没有给心脏供应足够的血液和氧气，进而导致心肌受损。

Metastases to the myocardium are less common, but arise more commonly when cancer spreads via the blood.

然而转移到心肌并不是很常见，但是当癌细胞传播到血液中时，心肌癌的可能性将会升高。

Now, with coronary artery disease, there's ischemia, or reduced blood flow to the region of myocardium supplied by that artery.

现在，冠状动脉性疾病患者存在心肌缺血，即由某一动脉供血的心肌血供减少了。

The signals are then passed onto fascicular branches, and spread through millions of Purkinje fibres over the ventricular myocardium.

这些信号随后传递到束状分支上，并通过心室心肌上数以百万计的浦肯野纤维传播。

Over time, this results in maximal vasodilation beyond the obstruction, and it helps the collateral vessel redivert blood to the ischemic myocardium.

随着时间的推移，这会使阻塞远端的血管最大程度地扩张，并帮助侧支循环将血流引向缺血的心肌。

If left untreated, however, the full thickness of the region of myocardium supplied by the occluded vessel may die.

然而，如若不治疗，堵塞血管供血的心肌区域全厚层将死亡。

They may relate to abnormal electrical events at the margins between ischemic and adjacent normal regions of myocardium.

它们可能与心肌缺血区和邻近正常区边缘的异常电活动有关。

We will now examine what happens when the full thickness of myocardium under lead v6 is dead.

我们现在将检查V6导联下，心肌全层死亡时的情况。

The end result is that blood is diverted, or stolen, away from the ischemic myocardium to non-ischemic areas, which further worsens the ischemia.

最终结果就是血液从缺血的心肌被转移或者说被“偷”到未缺血区域，进一步加剧了缺血。